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slc12a1 nkcc2 (Proteintech)

Name: slc12a1 nkcc2
Brand: Proteintech
Rating: 93 (36 reviews)

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Proteintech slc12a1 nkcc2
Slc12a1 Nkcc2, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 36 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 36 article reviews

slc12a1 nkcc2 - by Bioz Stars, 2026-02

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Flcn deletion causes cyst formation in the nephron, most prominent in the loop of Henle (LoH). A and B: Hematoxylin and eosin (H&E) staining of postnatal day 0 kidneys from control ( Flcn flox/flox ) and Flcn knockout (KO) ( Six2Cre ; Flcn flox/flox ) mice showing numerous cysts in both cortex and medulla. All KO mice ( N = 29) exhibited cystogenesis; representative images are presented. C and D: Immunofluorescence staining against LTL (proximal tubule), SLC12A1 (LoH), and WT1 (glomerular podocyte) confirming the presence of many cysts in the Flcn KO nephrons but not in controls. E and F: Higher magnification of C and D . White asterisks: Bowman's space; yellow asterisks: proximal tubule. G and H: Immunofluorescence staining against CDH1 SLC12A1 showing dilated LoH ( yellow arrows ). The lower left panels are higher magnification of the boxed regions , showing the irregularly curved lumen ( arrowheads ) with enlarged surrounding epithelia (SLC12A1 + /CDH1 + ) in Flcn KO but not in control LoH. The collecting ducts stained only with CDH1 are not dilated in either control or KO samples. Scale bars: 1 mm ( A and B ); 500 μm ( C and D ); 100 μm ( E–H ).

Journal: The American Journal of Pathology

Article Title: Folliculin Deletion in the Mouse Kidney Results in Cystogenesis of the Loops of Henle via Aberrant TFEB Activation

doi: 10.1016/j.ajpath.2025.05.010

Figure Lengend Snippet: Flcn deletion causes cyst formation in the nephron, most prominent in the loop of Henle (LoH). A and B: Hematoxylin and eosin (H&E) staining of postnatal day 0 kidneys from control ( Flcn flox/flox ) and Flcn knockout (KO) ( Six2Cre ; Flcn flox/flox ) mice showing numerous cysts in both cortex and medulla. All KO mice ( N = 29) exhibited cystogenesis; representative images are presented. C and D: Immunofluorescence staining against LTL (proximal tubule), SLC12A1 (LoH), and WT1 (glomerular podocyte) confirming the presence of many cysts in the Flcn KO nephrons but not in controls. E and F: Higher magnification of C and D . White asterisks: Bowman's space; yellow asterisks: proximal tubule. G and H: Immunofluorescence staining against CDH1 SLC12A1 showing dilated LoH ( yellow arrows ). The lower left panels are higher magnification of the boxed regions , showing the irregularly curved lumen ( arrowheads ) with enlarged surrounding epithelia (SLC12A1 + /CDH1 + ) in Flcn KO but not in control LoH. The collecting ducts stained only with CDH1 are not dilated in either control or KO samples. Scale bars: 1 mm ( A and B ); 500 μm ( C and D ); 100 μm ( E–H ).

Article Snippet: The following primary antibodies were used: rabbit anti-SIX2 (#11562-1-AP; 1:400 dilution; Proteintech, Rosemont, IL), rat anti-KRT8 (1:10; Developmental Studies Hybridoma Bank, Iowa City, IA), mouse anti–E-cadherin (CDH1) (#610181; 1:100; BD, Franklin Lakes, NJ); rabbit anti-NKCC2 (SLC12A1) (#SPC-401D; 1:100; StressMarq Biosciences, Victoria, BC, Canada), goat anti-NKCC2 (SLC12A1) (#PAS-142445; 1:100; Invitrogen, Carlsbad, CA), rabbit anti-phospho-S6 ribosomal protein (Ser 240/244) (#5364; 1:100; Cell Signaling Technology, Danvers, MA), rabbit anti-WT1 (#GR 3270281-4; 1:100; Abcam, Cambridge, UK,), LTL (#B-132; 1:200; Vector Laboratories, Newark, CA), guinea pig anti-NEPHRIN (#GP-N2, 1:200; PROGEN, Heidelberg, Germany), rabbit anti-TFE3 (#HPA023881; 1:100; Sigma-Aldrich, St. Louis, MN), and rabbit anti-TFEB (#A303-673A, lot numbers 8 and 9; Bethyl Laboratories, Montgomery, TX).

Techniques: Staining, Control, Knock-Out, Immunofluorescence

Uniform Manifold Approximation and Projection for Dimension Reduction plots of representative up-regulated genes in Flcn knockout (KO) loop of Henle (LoH). Uniform Manifold Approximation and Projection for Dimension Reduction plots and violin plots of representative up-regulated genes in the KO LoH. Atp6v0d2 , Rab27a , Lamtor1 , R r a gd , and Fnip2 are up-regulated in the homozygous KO LoH. Slc12a1 is used as an unaffected marker for LoH. Cluster numbers are presented in A. DT, distal tubule; NP, nephron progenitor; Pod, glomerular podocyte; PT, proximal tubule.

Journal: The American Journal of Pathology

Article Title: Folliculin Deletion in the Mouse Kidney Results in Cystogenesis of the Loops of Henle via Aberrant TFEB Activation

doi: 10.1016/j.ajpath.2025.05.010

Figure Lengend Snippet: Uniform Manifold Approximation and Projection for Dimension Reduction plots of representative up-regulated genes in Flcn knockout (KO) loop of Henle (LoH). Uniform Manifold Approximation and Projection for Dimension Reduction plots and violin plots of representative up-regulated genes in the KO LoH. Atp6v0d2 , Rab27a , Lamtor1 , R r a gd , and Fnip2 are up-regulated in the homozygous KO LoH. Slc12a1 is used as an unaffected marker for LoH. Cluster numbers are presented in A. DT, distal tubule; NP, nephron progenitor; Pod, glomerular podocyte; PT, proximal tubule.

Techniques: Knock-Out, Marker

Transcription factor E3 (TFE3) and mammalian target of rapamycin complex 1 (mTORC1) are activated in Flcn knockout (KO) nephrons. A–D: In situ hybridization of Atp6v0d2 and Rab27a in Flcn flox/flox and KO kidneys. Slc12a1 is used as a marker for loop of Henle (LoH). The right panels are higher magnification of the boxed regions in the left panels . E–H: Immunofluorescence staining for TFE3, SLC12A1 (LoH), and LTL (proximal tubule) showing TFE3 nuclear reactivity in the cortex ( E and F ) but not in the medulla ( G and H ). Nuclear localization of TFE3 is detected in glomerular podocytes, Bowman's capsule ( white arrowheads ), and proximal tubules ( yellow arrowheads ) in Flcn KOs but not in LoH. The right panels are higher magnification of the boxed regions in the left panels . I–L: Immunofluorescence staining of phosphorylated S6 ribosomal protein (pS6), SLC12A1 (LoH), and LTL (proximal tubule) showing the increased pS6 in the LoH ( yellow arrows ) of Flcn KO kidneys. The right panels are higher magnification of the boxed regions in the left panels . Scale bars: 500 μm ( A–D ); 100 μm ( E–L ).

Journal: The American Journal of Pathology

Article Title: Folliculin Deletion in the Mouse Kidney Results in Cystogenesis of the Loops of Henle via Aberrant TFEB Activation

doi: 10.1016/j.ajpath.2025.05.010

Figure Lengend Snippet: Transcription factor E3 (TFE3) and mammalian target of rapamycin complex 1 (mTORC1) are activated in Flcn knockout (KO) nephrons. A–D: In situ hybridization of Atp6v0d2 and Rab27a in Flcn flox/flox and KO kidneys. Slc12a1 is used as a marker for loop of Henle (LoH). The right panels are higher magnification of the boxed regions in the left panels . E–H: Immunofluorescence staining for TFE3, SLC12A1 (LoH), and LTL (proximal tubule) showing TFE3 nuclear reactivity in the cortex ( E and F ) but not in the medulla ( G and H ). Nuclear localization of TFE3 is detected in glomerular podocytes, Bowman's capsule ( white arrowheads ), and proximal tubules ( yellow arrowheads ) in Flcn KOs but not in LoH. The right panels are higher magnification of the boxed regions in the left panels . I–L: Immunofluorescence staining of phosphorylated S6 ribosomal protein (pS6), SLC12A1 (LoH), and LTL (proximal tubule) showing the increased pS6 in the LoH ( yellow arrows ) of Flcn KO kidneys. The right panels are higher magnification of the boxed regions in the left panels . Scale bars: 500 μm ( A–D ); 100 μm ( E–L ).

Techniques: Knock-Out, In Situ Hybridization, Marker, Immunofluorescence, Staining

Transcription factor EB (TFEB) is activated in Flcn knockout (KO) nephrons. A-D: Immunofluorescence staining for TFEB, SLC12A1 [loop of Henle (LoH)], and LTL (proximal tubule) showing TFEB nuclear reactivity in the cortex ( A and B ) and medulla ( C and D ) at postnatal day 0. Nuclear localization of TFEB is detected in proximal tubules ( yellow arrowheads ) and LoH ( yellow arrows ) in Flcn KOs. White arrowheads : Bowman’s capsule. The right panels are higher magnification of the boxed regions in the left panels . E and F: Immunofluorescence staining for TFEB, SLC12A1 (LoH), and KRT8 (collecting duct). The TFEB signal in the collecting duct remains unchanged. The right panels are higher magnification of the boxed regions in the left panels . Scale bars = 100 μm ( A–F ).

Journal: The American Journal of Pathology

Article Title: Folliculin Deletion in the Mouse Kidney Results in Cystogenesis of the Loops of Henle via Aberrant TFEB Activation

doi: 10.1016/j.ajpath.2025.05.010

Figure Lengend Snippet: Transcription factor EB (TFEB) is activated in Flcn knockout (KO) nephrons. A-D: Immunofluorescence staining for TFEB, SLC12A1 [loop of Henle (LoH)], and LTL (proximal tubule) showing TFEB nuclear reactivity in the cortex ( A and B ) and medulla ( C and D ) at postnatal day 0. Nuclear localization of TFEB is detected in proximal tubules ( yellow arrowheads ) and LoH ( yellow arrows ) in Flcn KOs. White arrowheads : Bowman’s capsule. The right panels are higher magnification of the boxed regions in the left panels . E and F: Immunofluorescence staining for TFEB, SLC12A1 (LoH), and KRT8 (collecting duct). The TFEB signal in the collecting duct remains unchanged. The right panels are higher magnification of the boxed regions in the left panels . Scale bars = 100 μm ( A–F ).

Techniques: Knock-Out, Immunofluorescence, Staining

Tfe3 deletion in the Flcn knockouts only ameliorates glomerular cysts. A: Scheme for the generation of Flcn ; Tfe3 double knockout (DKO) mice. Note that Tfe3 is located on the X chromosome. B and C: Immunofluorescence staining against TFE3, SLC12A1, and LTL showing the absence of TFE3 staining in the DKO mice. D and E: Hematoxylin and eosin (H&E) staining of postnatal day 0 kidneys from Flcn KO ( Six2Cre; Flcn flox/flox ) and Flcn ; Tfe3 DKO ( Six2Cre ; Flcn flox/flox ; Tfe3 del/Y ) mice showing amelioration of glomerular cysts in the DKO mice. Black arrows : Bowman’s space. Eleven DKO samples were examined; representative images are shown. F: Ratio of glomeruli with cysts to total glomeruli in H&E-stained sections. Sample numbers: 6 for Flcn KO; 8 for Flcn ; Tfe3 DKO. G–J: Immunofluorescence staining against LTL, SLC12A1, and WT1 highlighting proximal tubules, loop of Henle and glomerular podocytes. Glomerular cysts are reduced in DKOs ( yellow asterisks ), but cysts in the proximal tubules and loop of Henle are still present. The right panels are higher magnification of the boxed regions in the left panels . ∗ P < 0.05 ( P = 0.0340). Scale bars = 100 μm ( B – E and G– J ).

Journal: The American Journal of Pathology

Article Title: Folliculin Deletion in the Mouse Kidney Results in Cystogenesis of the Loops of Henle via Aberrant TFEB Activation

doi: 10.1016/j.ajpath.2025.05.010

Figure Lengend Snippet: Tfe3 deletion in the Flcn knockouts only ameliorates glomerular cysts. A: Scheme for the generation of Flcn ; Tfe3 double knockout (DKO) mice. Note that Tfe3 is located on the X chromosome. B and C: Immunofluorescence staining against TFE3, SLC12A1, and LTL showing the absence of TFE3 staining in the DKO mice. D and E: Hematoxylin and eosin (H&E) staining of postnatal day 0 kidneys from Flcn KO ( Six2Cre; Flcn flox/flox ) and Flcn ; Tfe3 DKO ( Six2Cre ; Flcn flox/flox ; Tfe3 del/Y ) mice showing amelioration of glomerular cysts in the DKO mice. Black arrows : Bowman’s space. Eleven DKO samples were examined; representative images are shown. F: Ratio of glomeruli with cysts to total glomeruli in H&E-stained sections. Sample numbers: 6 for Flcn KO; 8 for Flcn ; Tfe3 DKO. G–J: Immunofluorescence staining against LTL, SLC12A1, and WT1 highlighting proximal tubules, loop of Henle and glomerular podocytes. Glomerular cysts are reduced in DKOs ( yellow asterisks ), but cysts in the proximal tubules and loop of Henle are still present. The right panels are higher magnification of the boxed regions in the left panels . ∗ P < 0.05 ( P = 0.0340). Scale bars = 100 μm ( B – E and G– J ).

Techniques: Double Knockout, Immunofluorescence, Staining

Tfeb deletion in Flcn knockouts (KOs) restores all the phenotypes. A and B: Hematoxylin and eosin (H&E) staining of postnatal day 0 kidneys from Flcn KO and Flcn ; Tfeb double KO (DKO) mice showing reduction of cysts in both cortex and medulla ( black arrows ). Three DKO samples were examined; representative images are shown. C and D: Immunofluorescence staining showing the reduction of cysts in the DKO mice. Left panel: renal cortex; right panel: renal medulla. Yellow arrowheads : glomeruli; yellow asterisk: proximal tubule cyst; and yellow arrows: loop of Henle (LoH). Cysts are detected only in Flcn KO mice. E and F: Immunofluorescence staining against CDH1 and SLC12A1 showing amelioration of LoH abnormalities in the DKO. The right panels are higher magnification of the boxed regions in the left panels . G–J: Immunofluorescence staining of phosphorylated S6 ribosomal protein (pS6) showing its reduction in the DKO LoH. The right panels are higher magnification of the boxed regions in the left panels . Scale bars = 100 μm ( A–J ).

Journal: The American Journal of Pathology

Article Title: Folliculin Deletion in the Mouse Kidney Results in Cystogenesis of the Loops of Henle via Aberrant TFEB Activation

doi: 10.1016/j.ajpath.2025.05.010

Figure Lengend Snippet: Tfeb deletion in Flcn knockouts (KOs) restores all the phenotypes. A and B: Hematoxylin and eosin (H&E) staining of postnatal day 0 kidneys from Flcn KO and Flcn ; Tfeb double KO (DKO) mice showing reduction of cysts in both cortex and medulla ( black arrows ). Three DKO samples were examined; representative images are shown. C and D: Immunofluorescence staining showing the reduction of cysts in the DKO mice. Left panel: renal cortex; right panel: renal medulla. Yellow arrowheads : glomeruli; yellow asterisk: proximal tubule cyst; and yellow arrows: loop of Henle (LoH). Cysts are detected only in Flcn KO mice. E and F: Immunofluorescence staining against CDH1 and SLC12A1 showing amelioration of LoH abnormalities in the DKO. The right panels are higher magnification of the boxed regions in the left panels . G–J: Immunofluorescence staining of phosphorylated S6 ribosomal protein (pS6) showing its reduction in the DKO LoH. The right panels are higher magnification of the boxed regions in the left panels . Scale bars = 100 μm ( A–J ).

Techniques: Staining, Immunofluorescence

A. Single cell RNA-sequencing of two organoids samples of day 21 organoids with RSPO added at day 7 or day 14, integrated and plotted in the first two UMAP dimensions grouped as 4 main clusters. B. Expression distribution of key gene markers of distal nephron and ureteric epithelium. C. Re-analysis of the epithelial cluster (cluster 0) plotted in the first two UMAP dimensions, grouped as 4 main clusters. D. Expression of key markers show populations of early distal nephron (GATA3, CDH2, FGF8), Loop of Henle (IRX1, FXYD2, SLC12A1) and other distal nephron (KCNJ1, DEFB1, TMEM52B) markers. E. Kidney cell classification tool DevKidCC identified the lineage identities of cells in our organoid datasets in comparison to that of Mae et al , highlighting a difference in the epithelium present being distal nephron in our datasets while being ureteric epithelial-like in Mae.

Journal: bioRxiv

Article Title: In vitro plasticity between ureteric epithelial and distal nephron identity and maturity is controlled by extracellular signals

doi: 10.1101/2025.06.03.657609

Figure Lengend Snippet: A. Single cell RNA-sequencing of two organoids samples of day 21 organoids with RSPO added at day 7 or day 14, integrated and plotted in the first two UMAP dimensions grouped as 4 main clusters. B. Expression distribution of key gene markers of distal nephron and ureteric epithelium. C. Re-analysis of the epithelial cluster (cluster 0) plotted in the first two UMAP dimensions, grouped as 4 main clusters. D. Expression of key markers show populations of early distal nephron (GATA3, CDH2, FGF8), Loop of Henle (IRX1, FXYD2, SLC12A1) and other distal nephron (KCNJ1, DEFB1, TMEM52B) markers. E. Kidney cell classification tool DevKidCC identified the lineage identities of cells in our organoid datasets in comparison to that of Mae et al , highlighting a difference in the epithelium present being distal nephron in our datasets while being ureteric epithelial-like in Mae.

Article Snippet: The following primary antibodies were used for immunofluorescence at a concentration of 1:300: Goat polyclonal anti-GATA3 (Cat#AF2605; R&D Systems), Mouse monoclonal anti-KRT8 (Cat#AB115959; Abcam), Rabbit polyclonal anti-PAX2 (Cat#71-6000; Zymed Laboratories Inc.), Rat polyclonal anti-αPKC (Cat#sc-216; Santa Cruz), Rabbit monoclonal anti-GATA3 (Cat#5852; Cell Signalling Technology), Sheep polyclonal anti-NEPHRIN (Cat#AF4269; R&D Systems), Mouse monoclonal anti-E-CADHERIN (Cat#610181; BD Biosciences), Biotinylated Lotus tetragonolobus lectin (LTL) (Cat#B-1325; Vector Laboratories), Rabbit polyclonal anti-RFP (Cat#PM005; Medical & Biological Laboratories Co.), Goat polyclonal anti-LHX1 (Cat# sc-19341; Santa Cruz Biotechnology, Inc.), Mouse monoclonal anti-MES1/2/3 (Cat# 39795, ActiveMotif), Rabbit polyclonal anti-UMOD (Cat#BT-590; Biomedical Technologies Inc), Rabbit polyclonal SLC12A1 (Cat#18970-1-AP; Proteintech Group), Rabbit polyclonal SLC12A3 (Cat#LS-C662545; Sapphire Bioscience), Chicken polyclonal anti-GFP (Cat#ab13970; Abcam), Rabbit polyclonal anti-SIX2 (Cat#11562-1-AP; Proteintech Group), Rabbit polyclonal anti-UPK3A (Cat#HPA018415; Sigma-Aldrich), Mouse monoclonal anti-actin (ACTA2) (Cat#A2547; Sigma-Aldrich), Rabbit monoclonal anti-RET (Cat#3223, Cell Signalling Technology), Rabbit polyclonal anti-AQP2 (Cat#AB3274; Chemicon).

Techniques: RNA Sequencing, Expressing, Comparison

mRNA abundance of the main sodium transporters after CsA treatment. Competitive PCR analysis of ( A ) NHE3 in PT, ( B ) NHE3 in TAL, ( C ) NKCC2 in TAL and ( D ) NCC in DCT on rats treated with CsA for 21 days (black squares) or controls (grey circles). Data are reported as ratio relative to control with individual points (mean ± standard error of the mean). * P -value <.05; ** P -value <.01; *** P -value <.001.

Journal: Nephrology Dialysis Transplantation

Article Title: Cyclosporin-induced hypertension is associated with the up-regulation of Na + -K + -2Cl − cotransporter (NKCC2)

doi: 10.1093/ndt/gfad161

Figure Lengend Snippet: mRNA abundance of the main sodium transporters after CsA treatment. Competitive PCR analysis of ( A ) NHE3 in PT, ( B ) NHE3 in TAL, ( C ) NKCC2 in TAL and ( D ) NCC in DCT on rats treated with CsA for 21 days (black squares) or controls (grey circles). Data are reported as ratio relative to control with individual points (mean ± standard error of the mean). * P -value <.05; ** P -value <.01; *** P -value <.001.

Article Snippet: The membranes were probed with 1:1000 diluted antibodies against NHE3 and NKCC2 (OriGene, Rockville, MD, USA).

Techniques: Control

Immunoblotting of NHE3 and NKCC2 in renal CTX/OSOM and ISOM dissected from control (white bar) or CsA-treated rats (black bar). Equal loading was assessed by Coomassie Blue staining. Data are reported as ratio relative to control (mean ± standard deviation). * P -value <.05; ** P -value <.01.

Journal: Nephrology Dialysis Transplantation

Article Title: Cyclosporin-induced hypertension is associated with the up-regulation of Na + -K + -2Cl − cotransporter (NKCC2)

doi: 10.1093/ndt/gfad161

Figure Lengend Snippet: Immunoblotting of NHE3 and NKCC2 in renal CTX/OSOM and ISOM dissected from control (white bar) or CsA-treated rats (black bar). Equal loading was assessed by Coomassie Blue staining. Data are reported as ratio relative to control (mean ± standard deviation). * P -value <.05; ** P -value <.01.

Article Snippet: The membranes were probed with 1:1000 diluted antibodies against NHE3 and NKCC2 (OriGene, Rockville, MD, USA).

Techniques: Western Blot, Control, Staining, Standard Deviation

Furosemide challenge in kidney-transplanted patients and healthy subjects. ( A ) Immunoblotting of urinary NKCC2 expression in CsA-treated (CsA, gray dashed bar, n = 3) or FK506-treated patients (FK506, gray bar, n = 3) and healthy control subjects (Ctr, white bar, n = 3). Values are reported as ratio relative to healthy control. ( B ) Time course changes in FeNa + , ( C ) FeCl − and ( D ) FeCa 2+ in kidney-transplanted patients receiving CsA (CsA, black line, n = 3) or FK506 (grey dashed line, n = 3) and healthy control subjects (Ctr, grey line, n = 3) challenged with 50 mg furosemide per os. Values are reported as mean ± standard error of the mean. A * compares Ctr vs CsA; # compares CsA vs FK506 (one-way ANOVA). (D) *** compares Ctr vs CsA; ### compares CsA vs FK506 (one-way ANOVA). P -values: * and # P -value <.05; ** and ## P -value <.01; *** and ### P -value < .001.

Journal: Nephrology Dialysis Transplantation

Article Title: Cyclosporin-induced hypertension is associated with the up-regulation of Na + -K + -2Cl − cotransporter (NKCC2)

doi: 10.1093/ndt/gfad161

Figure Lengend Snippet: Furosemide challenge in kidney-transplanted patients and healthy subjects. ( A ) Immunoblotting of urinary NKCC2 expression in CsA-treated (CsA, gray dashed bar, n = 3) or FK506-treated patients (FK506, gray bar, n = 3) and healthy control subjects (Ctr, white bar, n = 3). Values are reported as ratio relative to healthy control. ( B ) Time course changes in FeNa + , ( C ) FeCl − and ( D ) FeCa 2+ in kidney-transplanted patients receiving CsA (CsA, black line, n = 3) or FK506 (grey dashed line, n = 3) and healthy control subjects (Ctr, grey line, n = 3) challenged with 50 mg furosemide per os. Values are reported as mean ± standard error of the mean. A * compares Ctr vs CsA; # compares CsA vs FK506 (one-way ANOVA). (D) *** compares Ctr vs CsA; ### compares CsA vs FK506 (one-way ANOVA). P -values: * and # P -value <.05; ** and ## P -value <.01; *** and ### P -value < .001.

Article Snippet: The membranes were probed with 1:1000 diluted antibodies against NHE3 and NKCC2 (OriGene, Rockville, MD, USA).

Techniques: Western Blot, Expressing, Control

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